A magnetic method of killing cancer cells has been developed by scientists in South Korea.

The technique uses a magnetic field to flip a “self-destruct” switch in tumours.

Researchers have demonstrated that the process works in bowel cancer cells and living laboratory fish. Programmed cell death, or apoptosis, is one of the body’s ways of getting rid of old, faulty or infected cells.

In response to certain signals, the doomed cell shrinks and breaks into fragments. These are then engulfed and consumed by amoeba-like immune cells.

Often in cancer, apoptosis fails and cells are allowed to keep dividing uncontrollably.

The magnetic therapy involves creating tiny iron nanoparticles attached to antibodies which bind to “receptor” molecules on tumour cells. When the magnetic field is applied, the molecules cluster together, automatically triggering the “death signal” that sets off apoptosis.

In laboratory experiments, bowel cancer cells were exposed to the nanoparticles and placed between two magnets. The cells were designed to light up green to signal that apoptopic clustering was taking place.

More than half the exposed cells were destroyed by magnetic activation, whereas no untreated cells were affected. In another experiment, magnetically-induced apoptosis in zebra fish caused the creatures to grow abnormal tails.

Details of the research, led by Professor Jinwoo Cheon, from Yonsei University in Seoul, appear in the journal Nature Materials.

The scientists wrote: “We have demonstrated that apoptosis signalling can be turned on in-vitro (in the laboratory) and in a zebra fish in-vivo (living) model by using a magnetic switch. Our magnetic switch may be broadly applicable to any type of surface membrane receptors that exhibit cellular functions on clustering.”

SIGN UP FOR THE BIOMAGNETISM ONLINE TRAINING COURSE BY CLICKING ON THE BELOW PAYPAL PAYMENT BUTTON

Detailed information about a person’s genetic makeup and their environmental risk factors does not change their disease risk prediction, according to the results of a new simulation study.

The researchers, from the Harvard School of Public Health, said that much more research is indeed needed before information on patients’ genetic variants could actually help doctors prevent or treat conditions.

“Our findings suggest that the potential complexity of genetic and environmental factors related to disease will have to be understood on a much larger scale than we initially expected to be useful for risk prediction,” study author Hugues Aschard, a research fellow in the epidemiology department, said in a Harvard news release. “The road to genetic risk prediction, if it exists, is likely to be long”.

The investigators examined whether or not disease risk prediction for diseases such as breast cancer, type 2 diabetes and rheumatoid arthritis would improve if environmental risk factors were considered along with genetic risk. The authors called this interplay of genetic and environmental factors a “synergistic effect.”

The researchers simulated a wide range of interactions between environmental risk factors and common genetic risk markers related to the three diseases to determine if this simulation model would improve risk prediction.

These disease models, however, showed no significant improvement in risk prediction, and the researchers concluded that with this method, risk prediction sensitivity would improve by no more than 1 to 3 percent.

Study author Peter Kraft, an associate professor of epidemiology at the Harvard School of Health, added: “For most people, your doctor’s advice before seeing your genetic test for a particular disease will be exactly the same as after seeing your tests.”

Comment
A few years ago, official medicine tried to convince us that genome research was going to be the panacea for almost everything, which was a mechanistic and materialistic way of understanding life. Now they realize that it cannot predict almost anything.

We were told in the past that we die with the same genes that we are born with. However, it is getting increasingly clear, especially through studies of epigenetics, that the human mind and free will have, along with environmental and other factors, a great power to shape and change our genes AND THE GENES OF FUTURE GENERATIONS.

Our genes influence our personality, but the way we live and act also influence our genes.

This is just another example of how “science” is supporting more and more every day what ancient traditions have been holding for ages.

How longer will we passively accept what they are telling us, simply because they are supposed to be very “scientific”?

When will start a real debate on what should be considered “scientific”?

SIGN UP FOR THE BIOMAGNETISM ONLINE TRAINING COURSE BY CLICKING ON THE BELOW PAYPAL PAYMENT BUTTON

Researchers say they’ve linked type 2 diabetes with earlier development of precancerous colon lesions and they recommend people with the blood sugar disorder start colorectal screenings at a younger age than others.

“Based on our data, it implies people with diabetes should get screenings earlier, possibly at age 40, rather than at age 50,” said Dr. Hongha Vu, a clinical gastroenterology fellow at Washington University in St. Louis.

Experts know that diabetes is linked with an increased risk of colon and other cancers. Vu’s team set out to determine if people with diabetes develop precancerous lesions, also called polyps or adenomas, earlier than people without diabetes.

The researchers compared the incidence of polyps in three groups: those 40 to 49 with and without diabetes and those 50 to 59 without diabetes. Each group had 125 people.

All had colonoscopies between June 2005 and June 2011. In a colonoscopy, doctors examine the large intestine with a long, thin tube that has a camera at the end. Any polyps found are removed so cancer can’t progress.

The younger men and women with diabetes had a rate of polyps similar to the older people without diabetes.

“We found that between the three groups, the adenoma detection rate in those 40 to 49 without diabetes was 14.4 percent, and it was significantly higher in those with diabetes in the same age range — at 30.4 percent”. “This is a similar rate as those 50 to 59 without diabetes.” The 50- to 59-year-olds had a rate of 32 percent.

Because diabetes cases are expected to soar in coming decades, partly driven by the obesity epidemic, the researchers believe the findings have important public health implications.
Without insurance, a colonoscopy costs about $1,200 or more.

Comment
There are bacteria that can cause diabetes and may also be involved in colon cancer as well.
For example Enterobacter cloacae, Salmonella typhus and many other.
The Biomagnetic Pair proposes to have the patient completely scanned to avoid these problems.

SIGN UP FOR THE BIOMAGNETISM ONLINE TRAINING COURSE BY CLICKING ON THE BELOW PAYPAL PAYMENT BUTTON

A new study is the first to show a direct link between a high body-mass index and the risk of developing heart disease, according to British and Danish researchers.

Body-mass index, or BMI, is a measurement based on height and weight. People with a BMI of 18.5 to 24.9 are normal weight while those with a BMI of 30 or more are obese. Those in between are deemed overweight.

For the study, the researchers analyzed data from more than 75,000 people and found that those with a high BMI had a 26 percent increased risk of developing heart disease. Further analysis using genetic and other data showed that a BMI increase of four points increases the risk of heart disease by no less than 52 percent.

“By doing epidemiological studies combined with genetic analysis, we have been able to show in a group of nearly 76,000 persons that a high BMI is enough in itself to damage the heart,” Borge Nordestgaard, chief physician at Copenhagen University Hospital, said in a university news release.

“Studies have also suggested a relationship between heart disease and obesity, but that is not enough to prove any direct correlation. Obese people can share characteristics or lifestyle traits that have an influence on both the heart and weight. Or there can be a reverse causality, that is, if it is the diseased heart that causes obesity and not the other way round,” said Nordestgaard, who is also a clinical professor in the health and medical sciences faculty at the university.

Study co-author Dr. Nicholas Timpson, a lecturer in genetic epidemiology at the University of Bristol in England, noted in the news release: “In light of rising obesity levels, these findings are crucial to improving public health. Our research shows that shifting to a lifestyle that promotes a lower BMI will reduce the odds of developing the disease.”

Comment
The Biomagnetic Pair technique has been saying for a long time that there are pathogens that contribute both to the development of heart disease and obesity, such as streptococcus or staphylococcus bacteria.
But the best thing is that they can be easily eliminated with this therapy.

SIGN UP FOR THE BIOMAGNETISM ONLINE TRAINING COURSE BY CLICKING ON THE BELOW PAYPAL PAYMENT BUTTON

SIGN UP FOR THE BIOMAGNETISM ONLINE TRAINING COURSE BY CLICKING ON THE BELOW PAYPAL PAYMENT BUTTON